Chernobyl AIDS & Fukushima AIDS
The ongoing global spread of radionuclides from the destroyed Fukushima Daiichi nuclear power plant is causing immune system-related illnesses in Japan, the USA, and worldwide. The same phenomenon occurred and is still occurring after the Chernobyl nuclear catastrophe in 1986, where it was termed “Chernobyl AIDS.”
One result of many studies conducted during the last few years in Ukraine, Belarus, and Russia is the clear finding that Chernobyl radiation suppresses immunity – a person’s or organism’s natural protective system against infection and most diseases.
The lymphatic system – the bone marrow, thymus, spleen, lymph nodes, and Peyer’s patches – has been impacted by both large and small doses of ionizing radiation from the Chernobyl fallout. As a result, the quantity and activity of various groups of lymphocytes and thus the production of antibodies, including various immunoglobulins, stem cells, and thrombocytes, are altered. The ultimate consequences of destruction of the immune system is immunodeficiency and an increase in the frequency and seriousness of acute and chronic diseases and infections, as is widely observed in the Chernobyl-irradiated territories… The suppression of immunity as a result of this radioactive contamination is known as “Chernobyl AIDS.”…
Apparently, impaired immunity triggered by Chernobyl radionuclides adversely affected all of the individuals, without exception, who were subjected to any additional radiation. (Yablokov et al., p. 87 & 92, emphasis mine)
So everyone who was exposed to Chernobyl radiation had their immune systems affected from the fallout. The same will happen to everyone exposed to Fukushima radiation, which is or will be the entire populations of Japan and the USA. This is known as “Fukushima AIDS.”
Why I am susceptible
I have a host of previously existing autoimmune disorders, which were caused by fetal exposure to radiation from the Castle Bravo nuclear bomb in 1954. These illnesses includes psoriatic arthritis, chronic fatigue, inflammatory bowel disease, irritable bowel disorder, and immune thrombocytopenic purpura. Everything has gotten much worse after Fukushima, especially since November 2011. So I wanted to know what the hell was going on, so I could treat and survive this thing. I have extreme sensitivity to many medications, with overblown side effects, so running to doctors and sheep-like obedience to them is out of the question. The medications which might be prescribed like TNF-alpha blockers would likely have life-threatening side effects.
T-lymphocytes are used in the body’s defense against pathogens. They arise in the bone marrow, and travel to the thymus, where they are differentiated into different kinds of T-cells. T-helper cells include Th1, Th2 and Th17 cells. T-regulatory cells (Treg) suppress the T-helper cells and bring the system into homeostatic balance.
T-lymphocytes are part of the body’s immune system; they recognise pathogens (foreign bacteria) and they produce cytokines – hormonal messengers that are responsible for biological effects in the immune system. The cytokines fall into two groups: those that are pro-inflammatory, and those that are anti-inflammatory.
The helper T-cells (a type of T-lymphocyte) produce enormous amounts of two types of cytokines: Th1 and Th2. The Th1 cytokines produced by the helper T-cells produce a pro-inflammatory response; The TH2 cytokines produce an anti-inflammatory response, but promote allergic responses…
The Th1-type cytokines produce inflammation to kill intracellular parasites (viruses and certain bacteria, such as Listeria and Mycobacterium tuberculosis – the bacillus that causes TB). These cytokines also perpetuate any form of autoimmune response, and can cause cell-mediated allergies.
Th1-type lymphokines are involved in the development of organ-specific autoimmune diseases, such as autoimmune uveitis, allergic encephalomyelitis, or insulin-dependent diabetes mellitus…
The TH2 cytokines counteract the effects of the TH1 cytokines – they have an anti-inflammatory action. But they also help kill extracellular pathogens (which live outside the body’s cells and are exposed to antibodies in blood and other body fluids).
The TH2 cytokines induce a pronounced allergic response. If you suffer from IgE-mediated allergies, or asthma, you are likely to be over-producing TH2-types of cytokines, and have a TH2-weighted imbalance.
Th2-cell predominance is found in patients with chronic graft-versus host disease, progressive systemic sclerosis, systemic lupus erythematosus, and allergic diseases. ( http://suite101.com/article/what-are-th1-and-th2-forms-of-immune-response-a118467 )
So T-cells arise from the bone marrow. Strontium-90 and plutonium are bone-seekers and collect in bones and bone marrow. Amounts of naive T-cells (Th0) were found to be reduced in Chernobyl. The thymus is the organ where these naive T-cells take on different characteristics, and become Th1 or Th2 cells, etc. The thymus was also affected in Chernobyl.
The Th1/Th2 axis
As above, there is a balance between Th1 and Th2. An imbalance means that a person is Th1-dominant or Th2-dominant. More Th1 means less Th2 and vice versa.
Th1-dominant people tends toward autoimmunity, they have rheumatoid arthritis, psoriasis, inflammatory bowel disorders, joint inflammation, multiple sclerosis. They are vulnerable to parasites.
Th2-dominant people have allergic reactions, allergic asthma, hives, rashes, sensitivity to household chemicals, lupus, atopic dermatitis etc. They are vulnerable to bacterial and viral infections.
It is obvious that I am highly Th1-dominant. I rarely have allergic reactions, but suffer from inflammation of joints and skin. So personally I am interested in herbs and medications that swing the balance towards Th2.
Th1 has to with cellular immunity, that is, it deals with pathogens like viruses and bacteria that are inside cells. Th2 has to do with humoral immunity, pathogens in the interstitial fluid between cells, like parasites.
Th17 cells were discovered in 2005. These cells are pro-inflammatory and cause inflammation in joints and skin in arthritis and psoriasis. Treg cells are immune suppressors, they secrete cytokines that reduce the Th17 response, and keep things from going out of control. This is another axis like Th1/Th2. Treg reduces Th17. Too much Th17 and your immune-related symptoms are amplified. Too much Treg and your body cannot fight fungi, bacteria and cancer.
It is obvious that I have way too much Th17 and way too little Treg. So I am interested in substances that increase Treg, but reduce Th1.
An antigen like a bacteria, virus, fungus, or plutonium particle will interact with the body and create an antigen-presenting cell (APC). This will tell the naive T-cell to become a Th1, Th2, Th17, or Treg cell. Radiation in the environment can affect both the Th1/Th2 and Th17/Treg balances.
This video is very technical, but it has a good discussion about the role of interleukin-17 (IL-17) in inflammatory diseases (at 18 minutes in). This cytokine is involved with Th17 imbalance. At 23 minutes a case study is presented in which a baby was not able to produce Treg due to defective bone marrow. Around 33 minutes he talks about the role of Treg in gut biology, how the gut flora are affected by lack of Treg cells.
A word about Candida fungi… ordinary Th17 and Th1 fight fungi in the body, but in the case of Candida, Treg is converted into Th17, and kills the fungus. The Th17 goes away when the infection clears up, and little or no inflammation results. Therefore clearance of Candida is associated with less inflammation, not more as with other pathogens. Substances and herbs that promote Treg are important to fight Candida infections.
In Chernobyl, and sites of other nuclear catastrophes like Mayak, reduced amounts of T-cells were noted, along with polarization of T-cells along the two axes. The imbalances can go any of 4 ways, but it seems that more often Th1 is pushed up at the expense of Th2. I will get into this more in the next post.