Iodine-131 detections in Finland and Norway preceded by detections in Japan.

The STUK radiation protection agency of Finland announced Tuesday that iodine-131 had been detected in the Lapland area of northern Finland, and that I-131 also had been detected in Norway, between May 4-11.

Also, cesium-134, niobium-95, and cobalt-60 were found in southeast Finland. These two types of detections may or may not be related.

These detections were not caused by the Chernobyl forest fire, since I-131 decays away in 80 days, and Cs-134 decays away in 20 years. The Chernobyl catastrophe occurred in 1986, 29 years ago.

Iodine-131 levels in Gunma prefecture sludge skyrocketed on their May 1 report to the highest readings since the huge Fukushima emissions of July 2013. These analyses were actually done in April 21-23. This area is to the southwest of the Fukushima plant.

Toyahashi sludge was also elevated on April 21. This area is to the west of Tokyo.

The lag time involved for the gaseous iodine to move from Japan to Scandinavia would allow the I-131 to decay by 50-75%, so the Finland and Norway detections are more robust than they look at first glance.

On May 12, I-131 was found in Chiba prefecture drinking water (not sludge). So it looks like the latest fission event is still going on.

I-131 was also (possibly) detected in river water in Avignon, France. But, as was explained here by the head of the CRIIRAD laboratory, this energy range measure also includes cesium and natural NORM radionuclides. They had to change the verbiage on their graphs due to this blog.

Chernobyl forest fire plume now on the US west coast.

The forest fire near Chernobyl that broke out a week ago has spread radionuclides, notably plutonium, over eastern Europe, Asia, Alaska, and is now moving in over the west coast of the US.

This image from the downloaded Windows HYSPLIT atmospheric dispersion model shows the spread of this plume, as of 2 PM Eastern time on May 7.

This simulation uses actual winds, not predicted winds.

It has moved over southern Alaska, where it is currently raining, and northern B.C. Some tendrils have moved over NW Washington, Salinas CA, Fresno CA, Santa Barbara CA, and Las Vegas NV.

For users of Google Earth or other visualization software, a KML file is available here.

As of Friday morning, the rain outside of Fresno has some measure of this smoke plume mixed in with it, and is falling as snow in the mountains to the northeast.

The plume should become entrained in a storm that is developing in this area. This image is a forecast for Saturday night. A snowstorm is moving over the Rockies, and the rain and snow will later move over the northern plains, the upper Great Lakes, into Canada.

Largest forest fire since 1992 endangers Chernobyl nuclear plant.

A forest fire has broken out in the Chernobyl exclusion zone, within 20 km of the nuclear plant.

(Sputnik) The forest fire is being fought by more than 200 firefighters, National Guard was put on high alert… A forest fire has erupted in the Chernobyl Exclusion Zone. More than 200 firefighters, 15 fire engines, two aircraft and one helicopter are battling the fire, according to Ukraine’s acting emergencies minister Zoran Shkiryak.

Ukraine’s Interior Minister Arsen Avakov wrote on his Facebook page that as of 6:30 PM Kiev time (3:30 PM GMT) the situation had gotten worse and the fire was approaching the Chernobyl nuclear plant.

“The forest fire is heading in the direction of Chernobyl’s installations. Treetop flames and strong gusts of wind have created a real danger of the fire spreading to an area within 20 kilometers of the power plant. There are about 400 hectares [988 acres] of forests in the endangered area. The Prime Minister has called an urgent session of the emergencies commission. National Guard and Interior Ministry forces have been put on combat alert”, Avakov’s statement reads…

According to the statement, Yatsenyuk has stressed that the authorities are tackling the situation despite the fact that the fire is the largest seen since 1992.

According to this article (Russian, Google translation):

Fire on the territory of special plant “Chornobyl Forest” can lead to secondary contamination by radioactive substances, said in comments RIA Novosti Ukrainian ecologist Vladimir Boreyko.

It is worth noting that according to the Ministry of Internal Affairs of Ukraine, in the exclusion zone of 400 hectares of forest burn. However, according to environmentalists, which is based on images from space, fire area exceeds 10 thousand hectares…

As stated in an interview with RT Deputy Coordinator “Environmental Watch on the North Caucasus,” Dmitry Shevchenko when burning wood is not only carbon dioxide emissions, and soot, which spreads over hundreds and thousands of kilometers. Fire around the Chernobyl zone, according to experts, is dangerous because the radionuclides contained in the wood, into the atmosphere, may fall anywhere and spread over a long distance.

Shevchenko said that the Chernobyl zone is especially important to preserve the forest, because they are naturally conservative radionuclides that accumulate in the soil and wood.

Ukrainian ecologist Vladimir Boreyko believes that the fire on the territory of special plant “Chornobyl Forest” can lead to secondary contamination by radioactive substances. “We need to figure out where it burns wood: where the radiation spot, or where these spots are not. If there are spots where there is, it’s just air pollution. If there are spots where it is, of course, that’s too bad, because it is a secondary contamination by radioactive substances “- quoted by RIA Novosti ecologist.”

It was discovered recently that trees did not decay in the highly contaminated Red Forest near Chernobyl.

Scientists who have been studying the environment inside the Zone of Alienation since 1991 noticed something about these trees, specifically what they described as “a significant accumulation of litter over time” in a study published recently in Oecologia. And by “significant,” they mean the trees are not decomposing and their leaves are just sitting there on the ground, not decomposing either. This is especially so in the Red Forest, an area of woodland around Chernobyl named thusly because the trees turned a ginger color and died due to the worst radiation poisoning in the area. In an interview with Smithsonian magazine, lead author of the study and biologist at the University of South Carolina Timothy Mousseau called all this non-decayed organic matter “striking, given that in the forests where I live, a fallen tree is mostly sawdust after a decade of lying on the ground.”

The reason for this lack of decay around Chernobyl is that microbes, bacteria, fungi, worms, insects, and other living organisms known as decomposers (because they feed on dead organisms) are just not there and not doing their jobs. Mousseau and his team discovered this after leaving 600 bags of leaves around Chernobyl in 2007. When they collected the bags in 2008, they found that the bags filled with leaves placed in areas with no radiation had decomposed by 70 to 90 percent, but the leaves in areas with radiation? They only decomposed about 40 percent. “There is growing concern that there could be a catastrophic fire in the coming years,” Mousseau told Smithsonian. (link)

Last week, smoke from massive Siberian fires was seen on satellite over the US east coast. Expect this plume to hit the US within a week.

No corium in Fukushima Unit 1 reactor vessel.

The muon scan of the Unit 1 reactor vessel at Fukushima has been completed. No nuclear fuel was fuel in it. No surprise.

According to this article at Simply Info,

TEPCO published the results of the muon scan done by IRID and their cooperating partners. No fuel was found in the reactor vessel of unit 1 Fukushima Daiichi… This was as expected by many (including us) who have been documenting the other evidence that indicated this was the likely case… Also noted on these images was the lack of detectable solid vessel in the bottom head area. The center lower section of the reactor vessel appears to be missing. The uniform dark outline of the reactor vessel is noticeably missing…

The corium has burned through the vessel and has melted through to the basemat and drywell floor. The question is whether it burned through that too, into the soil underneath. I would say this is very likely.

From this long Sandia pdf,

The drywell floor is subdivided into three regions (i.e., cavities) for the purposes of modeling molten-core/concrete interactions (see Figure 20). The first region, which receives core debris exiting the reactor vessel, corresponds to the reactor pedestal floor and sump areas (CAV 0).

Debris that accumulates in CAV 0 can flow out through a doorway in the pedestal wall to a second region representing a 90 degree sector of the drywell floor (CAV 1). If debris accumulates in this region to a sufficient depth, it can spread further around the annular drywell floor into the third region (CAV 2). This discrete representation of debris spreading is illustrated in Figure 20. (p. 64)

It was already assumed that the molten fuel had interacted with the concrete:

The three accidents (i.e., the accidents in Units 1, 2, and 3), while similar in many ways in terms of SBO accident progression, each proceeded to different degrees of core damage, with Unit 1 believed to be the most severely damaged of the three. It is believed that the Unit 1 core damage proceeded to the point of lower vessel head failure that released core materials to the containment cavity where core-concrete interactions likely initiated. Units 2 and 3 are believed to be less damaged. Collectively, the accidents likely reflect varying degrees of core/reactor damage and are therefore an invaluable source of information that can validate/confirm our current understanding of severe reactor accidents and provide new understanding not currently realized in our body of knowledge. (p. 18)

I was already talking about the corium-concrete reaction when I was still posting at the Japan Earthquake scribble. This must have been in early 2011. The corium-concrete reaction liberates all sorts of radionuclides that wouldn’t have been released otherwise, and the concrete particles help spread the radioactive particles into the wind.

Note high levels of Ba-140 and La-140 on March 15-16. Lanthanum-140 is the daughter product of Barium-140… If a TMI-style meltdown occurred, using Dr. Saji’s inventory figures (below), there would have been a 23:1 ratio of I-131 to Te-129m released. The Takasaki figures indicate a 1:2 ratio. The data do not support this scenario…

“This study describes the increase in iodine activity released to the atmosphere during a severe accident due to the radioactive decay of tellurium precursors… here it is seen that the iodine activity in the atmosphere is due disproportionately to I-132. Unlike the longer-lived isotopes, most I-132 (half-life of 2.30 hr.) existing early in the accident will decay before the significant atmospheric releases which follow reactor vessel failure. However, the supply is replenished by the decay of Te-132, which is released in large quantities from the drywell rubble.”

Takasaki is almost 100 miles from the Fukushima plant. Thus most of the I-132 which was released would be decayed. The large amounts of I-132 detected must have come from the decay of Te-132.

From p. 533, “Upon contact, the molten core material (the so-called “corium”) starts to react with the material of the basemat concrete… when the reaction zone is flooded with sump water… the highest temperatures might be reached… the molten-core – concrete interaction is the principal source of the release of the low-volatility fission products to the containment. The volatilization of these elements, such as barium, strontium, lanthanum, and cerium, is strongly supported by the gas bubbles which penetrate through the molten zone.”

Note again the high concentrations of barium and lanthanum, and that of tellurium.

Conclusion: The emissions observed at Takasaki were not due to a TMI-style accident, but one in which corium interacted with concrete and water. This released significant concentrations of barium, lanthanum, and strontium into the atmosphere.

Well, it’s about time they did this scan. It’s only been 4 years.

FUKU+4 HEALTH NOTE: Really nasty hand-foot-mouth rash on my hands and feet. This is from a disseminated enterovirus infection, from a virus I caught almost a year ago. I don’t know what strain or type of enterovirus it is. It has infected the neurons in the brain and spinal cord (thus it is myalgic encephalomyelitis). Minor improvement today… I did a technical thing, added a Mid-p feature to my Fisher’s exact test program for large samples. It is good to know that I can do math and scientific programming again. Things are still dire, though.

Fuku+4. Cancer rates start taking off.

The 4-year anniversary of the catastrophe at the Fukushima Daiichi is upon us. Radioactive emissions into the air and the Pacific Ocean continue, as they apparently will for centuries or millenia.

Radioactive contamination of air, water, and food is causing and will continue to cause cancer, heart disease, and all immune-related disease, including neurological disease, indefinitely.

But there is a latency period, or time lag, between initial radiation exposure and the development of these diseases. This document, issued by the World Trade Center Health Program, officially determines the minimum latency of types of cancers subsequent to the 9/11 disaster in 2001. This determination was stipulated by Congress after the passage of the James Zadroga 9/11 Health and Compensation Act of 2010.

The assessment of minimum latency periods for various types or categories of cancer is straightforward when exposures occur at a single point in time or regularly. However, most human exposures to carcinogens vary significantly over time, making a precise determination of minimum latency periods difficult… Therefore, the Administrator derived minimum latency estimates using several methods based on the best available scientific evidence for each type or category of cancer considered…

4B: Estimates of cancer latency obtained from statistical models used to estimate the lifetime risk of low-level ionizing radiation-related cancers.

The use of a radiation-induced cancer latency estimate is supported by scientific literature indicating shared mechanisms of carcinogenesis that apply to most solid tumors. Furthermore, cancers that may develop as a result of radiation exposure are indistinguishable from those that occur as a result of exposure to other carcinogens.

If multiple estimates of minimum latency based on statistical modeling in epidemiologic studies were available in the scientific literature, the Administrator’s policy is to resolve any uncertainties inherent in this method [Latency Method 4] in favor of the WTC Health Program member by selecting the shortest latency period…

For solid cancers as a group, an estimate of minimum latency of 4 years is available from statistical modeling of risk between exposure to low-level ionizing radiation and solid cancers [Latency Method 4B].

So cancer latency associated with 9/11 dust is the same as cancer latency associated with Fukushima radiation.

The Administrator has selected minimum latencies for the following five types or categories of cancer:

(1) Mesothelioma — 11 years;
(2) All solid cancers (other than mesothelioma, lymphoproliferative, thyroid, and childhood cancers) — 4 years;
(3) Lymphoproliferative and hematopoietic cancers (including all types of leukemia and lymphoma) — 0.4 years (146 days);
(4) Thyroid cancer — 2.5 years; and
(5) Childhood cancers (other than lymphoproliferative and hematopoietic cancers) — 1 year.

So leukemia and lymphoma started developing in some people a few months after 3/11/11. I’m thinking here of Kevin Blanch, who developed AML leukemia in this time frame… though there are many others.

All other childhood cancers (20 years old or less) started developing in March 2012.

Thyroid cancer in adults started developing in September 2013.

All solid cancers in adults start developing NOW.

Solid cancers are all cancers involving solid tumors like breast cancer, colon cancer, lung cancer, liver cancer, etc.

There is nothing hard and fast about this. Some people have developed cancer already, before this minimum latency period. But this is an official number that was developed as a result of a trade-off. These minimum latencies are in stark contrast to latencies of 5, 10, 20 years that are used in medical literature, and skew results to the pro-nuclear side.

But the Fuku+4 point is significant, not just for minimum latencies, but for the point when certain cancer rates start accelerating. Thyroid cancer in Chernobyl children started taking off at the Chernobyl+4 point.

Sickness in general, among Chernobyl liquidators and their children, also significantly rose at the Chernobyl+4 point. This is very simple, the subject is either sick (from anything), or not. The rise in the sickness rates of the children show that this phenomenon was not due to aging.

My research on myalgic encephalomyelitis.

I wanted to get this out there now. Big changes are coming… I have gotten much sicker after the cold weather started, and getting another virus infection. I am moving out of my home soon, and I don’t know how this is possible without significant muscle exertion, which will makes the disease much worse. So massive uncertainty clouds my immediate future.

I still think I will get better after the weather warms up, but right now there is more snow and severe cold in the forecast, and I don’t know when this will happen.

Most of the links are from Facebook. I apologize to those who cannot access it. There is a way to synchronize Facebook posts to this blog, but I am too sick to do it now.

Type I interferons and interferon-gamma in ME. (FB link)

Immunosuppression causes persistence of enteroviral infections in ME, is the foundation of exertion intolerance, and can be reversed by Type I interferon blockers. (FB link)

Enterovirus infections of the central nervous system. (FB link)

Enterovirus infection of the gastrocnemius muscle, IDO, and peroxynitrite. (FB link)

Serotonin, IDO, and reduced tryptophan catabolism in ME. (FB link)

Picolinic acid deficiency in ME and the kynurenine pathway. (FB link)

TGF-beta is released from muscles 24 hours after exercise, and reduces levels of interferon-gamma. (FB link)

TGF-beta reduces NK cell cytolytic activity and MHC I and II expression. (FB link)

Kynurenine as a possible treatment for ME. (FB link)

Dysregulation of the STAT-1 protein in ME. (FB link)

Activin in ME, generated from muscle activity. (FB link)

Caffeine inhibits tryptophan hydroxylase, which is upgraded after exercise. (FB link)

Swimming exercise increases serotonin levels, and decreases corticosterone, interferon-gamma, TNF-alpha, and IDO levels. (FB link)

The TGF-beta autocrine loop in ME. (FB link)

Psychedelics and the desensitization of the 5-HT2A receptor. (FB link)

Exercise-induced reduction of interferon-gamma levels is the cause of post-exertional immune exhaustion in ME. (FB link)

GABA-B positive allosteric modulators are an effective treatment for ME, but are not yet approved by the FDA. (FB link)

List of TGF-beta inhibitors. I have determined that only those medications that use the Smad pathway are effective for ME. (link)

Brief moderate stretching is effective for reducing post-exertional symptoms after exercise. (FB link)

Moderate acute exercise increases TGF-beta, alpha-amylase, and IgA 24 hours after exercise. (FB link)

Noise increases tryptophan hydroxylase levels, which is a factor in noise hypersensitivity in ME. (FB link)

Brown fat, a key player in ME. (FB link)

Follistatin increases muscle mass after exercise, reduces TGF-beta levels via the Smad pathway, and is a myostatin inhibitor. The muscle repair process recruits interferon-gamma from tissues, and is the cause of exertion intolerance. (FB link)

Interferon-gamma deficiency in “chronic fatigue syndrome” – 1990, after the name change from ME. (FB link)

Ionizing radiation exposure is part of the ME disease process, by its generation of peroxynitrite (ONOO-) via radiolysis. (FB link)

Destruction of groups 1 and 3 innate lymphoid cells and the resultant distortion of commensal bacteria is a fundamental aspect of the ME disease process. (FB link)

Psilocybin reduces the activity of the default mode network. The inability to deactivate the DMN is behind the overwhelming neurological and cognitive symptoms in severe ME. (FB link)

Actimmune (interferon-gamma) is an effective treatment for ME. It does not reduce Type I interferon levels, though, which is necessary for a cure. (FB link)

Psilocybin reduces DMN activity by activating the 5-HT2A receptors. The receptors are typically desensitized in ME due to excessive serotonin production. (FB link)

A high-fat diet restores interleukin-22, and also restores the balance of microbiota in the gut. (FB link)

Limbic kindling vs. central sensitization in ME. My response to Leonard Jason’s research. (link)

Ketogenic diet for M.E.: Can it really be this simple? (link)

How ME and radiation sickness has altered my mechanism of thinking. (link)

Origin of M.E. disease process in the immune system. I have found since this, that an enteroviral infection of certain immunoprivileged areas of the brain does the same thing. Any viral, radiotoxic, or other toxic infestation of these brain areas will cause this. It is now known that the upgrade of Type I interferons is behind the dysregulation of the JAK/STAT pathway. (link)

Radiation as the cause of my persistent viral and neurological illness.

I wrote this post about how my way of thinking had changed on July 22, 2014. I try to log everything that happens to me… “where and when” is always essential. The exact timing yields important clues.

I was touted as a “boy genius” as a child… but I am only moderately intelligent. My talent has always been a phenomenal memory. My memory has enabled me to visualize what is going on in large computer programs, and mathematical algorithms that I have developed. I was able to collate large amounts of seemingly disparate information and unify it into a whole. I always saw the “big picture” in front of me. I tried to utilize this in this blog.

Now my big picture vision is a tiny pinhole. I can still do easy tasks like Step 1, Step 2, etc… as long as they have no physical involvement, and I have absolute peace and quiet, with no interruptions.

At the time I wrote the post, I didn’t know how much of this was due to enteroviral ME or radiation exposure. Fukushima radiation has been affecting my memory. I remember in June 2013, when the last major cloud of radioactive iodine started moving across the US, and my thyroid started to swell again, I had a hard time completing simple tasks. I tried to replace a headlight lamp in my car… but I kept reaching for the wrong bulb. I had a heck of a time with it. Online memory tests indicated that I may have had Alzheimer’s. I was convinced that radiation had finally caused me to come down with Alzheimer’s disease. But this was “brain fog”, which is common with fibromyalgia, ME/CFS, SEID, etc.

After the severe enteroviral illness which started on April 1, 2014, encephalomyelitis symptoms started around April 20. The vision in my left eye started getting dark and blurry. Then nystagmus commenced in my left eye. I developed optic neuritis in the left eye, and Bell’s palsy, which is facial paralysis, on the left side of my face. It is common to have these kind of symptoms on one side of the body only with encephalitis and encephalomyelitis, and MS, which is a kind of encephalomyelitis.

In late May, I suddenly went completely deaf. Shortly after this, my hearing started creeping back in, but it was accompanied by very loud tinnitus. Loud noises started to bother me… soon it was any noise, however small. This is called hyperacusis.

A benign tumor had developed on my pituitary gland in 2011. This greatly increased symptoms of my autoimmune disease, and I suffered widespread inflammation. My thyroid in particular, was affected. In January 2012, the tumor had grown so large that affected the fluid transport in my right eye, and it started to bulge out. I lost vision in the eye, except for a small circular ring, and suffered severe migraines. Ololiuqui seeds, which providentially arrived in the mail at this very time, caused the tumor to shrink. I later found that these seeds contain hydergine (discovered by the great chemist Dr. Albert Hoffmann), and I started taking this medication in tablet form, which worked very well to shrink the tumor. Unfortunately, not all the pituitary functions had come back to normal.

So the first thing I thought when I developed tinnitus and hyperacusis, was that the tumor had come back. So I doubled my usual dose of hydergine. Within a half-hour, the tinnitus and hyperacusis had increased tremendously, and the elevated level of this lasted a week. I had researched this medication throroughly, and knew it had been banned in France, due to suspected effects on the heart. What happens is that hydergine binds with the 5-HT2B serotonin receptor, which then releases a large amount of the anti-inflammatory cytokine TGF-beta. This cytokine is implicated in heart injury. It also opposes interferon-gamma, and destroys immune cells which release this pro-inflammatory cytokine. IFN-gamma is the main cytokine responsible for the creation of the IDO enzyme. IDO dysfunction is implicated in many neurological diseases.

Well, this was coming on so fast, that by mid-June I had reached the point where more than one sound happening at once would make me freeze up. My brain could no longer process two things at once. My memory was completely gone, to where I could not remember one second ago. It was like my consciousness was completely erased. These bouts lasted for just a few minutes. But I became frantic. It was around this time that post-exertional malaise (PEM) started occurring. That means these bouts would occur 24-48 hours after any kind of exercise, going to the store, etc.

I had already come across the ICC, the International Consensus Criteria, and I knew I had myalgic encepahlomyelitis. I tried with great difficulty to find forums and FB groups, in order to talk to people who had this disease. But all I could find were people who lacked energy to do anything, who were housebound and bedbound in some cases, and who had “brain fog”. On a scale of 1 to 10, brain fog was 1, and what I had was a million.

It turns out that the disease these people have is not encephalomyelitis at all. It is now called SEID (Systemic Exertional Intolerance Disease). I have exertional intolerance too. A lot of diseases have this as a symptom. The problem was that the CDC changed the name of ME to “chronic fatigue syndrome” (even though it does not include chronic fatigue) in 1988. It also included the disease now called SEID. The name was changed again to ME/CFS in order that it would be “taken more seriously”. So the people with enteroviral encephalomyelitis, which ME traditionally referred to, were left out in the cold completely.

My research has now led me to understand that the exertion intolerance in true ME is due to immunosuppression. This is the shutdown of Th1 cellular immunity, and the lack of Th1 cells in the body. These produce interferon-gamma. This happens because, in certain important areas of the brain, cellular immunity causes destruction of neurons, and the body removes the inflammation completely, allowing the enterovirus to spread.

So there are two things going on. Severe IFN-gamma deficiency causes IDO dysfunction, and a greatly increased amount of serotonin and glutamate activity in the brain. This is metabolic, and in itself causes neurological symptoms. Also, these neurotransmitters bind to receptors, which are neurons infected with the virus. This is where encephalomyelitis in involved, and makes things unpredictably worse.

Muscles release TGF-beta 24 hours after exercise, which drives IFN-gamma and IDO down further. Muscle activity also causes enteroviruses to replicate. This is the cause of PEM.

So what does this have to do with radiation? Dr. Martin Pall has said that the main risk factors for getting ME are:

1. High levels of inflammation.
2. PPAR dysfunction (high blood pressure).
3. Abnormal cortisol levels.

I already had autoimmune disease before Fukushima. The pituitary tumor, as I noted, greatly increased the amount of inflammation. It also disturbed ACTH function by the pituitary, which produces cortisol.

Pituitary tumors occurred at elevated levels after Chernobyl in the affected areas. Iodine-131, cesium-137, strontium-90, and plutonium are implicated. My thyroid problems increased in late 2011 with the radioactive iodine cloud, which I believe were caused by the action of I-131 on the pituitary, not so much the thyroid itself.

The enterovirus caused a case of the “flu” which would have gone away in a week, without the pituitary dysfunction, and accumulation of radioactive toxins in my body. But instead the enterovirus was not cleared out, spread to the brain, and caused a lifelong neurological disease.

Yablokov has a section in his book (pp. 104-112) “Chernobyl: Consequences of the Catastrophe for People and the Environment” which is devoted to neurological illnesses of those contaminated by Chernobyl radiation.

Previous views claiming resistance of the nervous system to radiation damage are refuted by the mounting collective data that demonstrate nervous system illnesses among the populations of the contaminated territories, especially liquidators. Even rather small amounts of nuclear radiation, considered harmless by former measures of radiation protection, have resulted in marked organic damage. Clearly, the existing radiation levels in the contaminated territories have harmed the central nervous system of countless people.

For many inhabitants of the contaminated territories, especially persons that were radiated in utero and liquidators, nervous system functions, including perception, short-term memory, attention span, operative thinking, and dreaming, are deteriorating. These conditions are associated with deep cerebral hemispheric damage: diencephalic areas, deep frontal, and temporal lobes, and occipitoparietal parts of the cerebral hemisphere. Low-dose radiation damages the vegetative (autonomic) nervous system. The fact that intellectual retardation is found in 45% of children born to mothers who went through the Hiroshima and Nagasakai nuclear bombardment is a very troubling concern (Bulanova, 1996).

ME (myalgic encephalomyelitis) is not chronic fatigue syndrome or SEID.

This table indicates the rate of symptoms of 420 UK patients who were classified as having myalgic encephalomyelitis. This is from ‘Myalgic encephalomyelitis–a persistent enteroviral infection?’, a report published in 1990, shortly after Dr. Ramsay’s death. It was the last thing he did.

CFS and ME/CFS were introduced in 1988, after an outbreak of a fatiguing illness in the Lake Tahoe area. ME was already an established name for a particular disease. The new disease did not fit, so ME and the new disease were thrown together and conflated.

The Institute of Medicine has recently renamed ME/CFS to SEID, systemic exertion intolerance disease. The committee noted that ME/CFS did not feature myalgia (muscle pain), or symptoms of encephalomyelitis (inflammation of the brain and spinal cord).

Out of the 420 patients in the table, 383 had evidence of enteroviral infection. Only 3 had evidence of Epstein-Barr virus. ME is overwhelmingly an enteroviral disease.

Note that chronic fatigue is not even on the list. Muscle fatigue or weakness occurs in 100% of patients. Myalgia or muscle pain occurs in 80% of them. According to the IOM, all SEID patients have chronic fatigue, myalgia is not an important symptom, and muscle weakness is not mentioned.

Auditory phenomena occur in 69% of the patients. Tinnitus is very frequent, and deafness is mentioned. This is due to an infection of the central nervous system. Tinnitus is infrequent in SEID. Auditory phenomena occur at a rate three times that of orthostatic tachycardia in ME. POTS is much more infrequent than tinnitus.

Visual disturbances occur at a much higher rate than with SEID. Hyperaethesia, or sensory hypersensitivity, occurs at a very high rate.

It is clear that ME is an infection of the brain and spinal cord, which affects the muscles, and is an immune deficiency disease that allows the infection to persist. Enteroviruses spread by infecting neurons. High rates of neurotransmitter activity, primarily serotonin and glutamate, are brought on by the immune condition. The receptors that these neurotransmitters bind to are infected by the virus.

ME is a very serious disease. SEID was never ME, and the conflation of the two completely different diseases has brought harm to ME patients, and is continuing to do so.

The IOM’s renaming of ME/CFS to SEID is welcome for ME patients. And it is timely because there is currently an epidemic of enterovirus 68 that is infecting children in the US and Canada. Many of them may develop ME, not SEID.

I demand that all Facebook groups, ME associations, websites, blogs, and other entities that use the name ‘ME’ or ‘ME/CFS’ immediately change their names, unless they are devoted specifically and only to myalgic encephalomyelitis.

Cold weather and radiation.

With another bout of cold weather engulfing the US, it’s time to review a little meteorology and atmospheric chemistry. Enenews has posted an article that refers to how radiation increases in cold weather due to the air being compressed at the surface.

Our results illustrate that accidents… could have significant trans-boundary consequences. The risk estimate [shows] increased surface level concentrations of gaseous radionuclides in the Northern Hemisphere during winter and a larger geographical extent towards the north and the east… This is related to the relatively shallow boundary layer in winter that confines the emitted radioactivity to the lowest part of the atmosphere close to the surface…It is the view of the authors that it is imperative to assess the risks from the atmospheric dispersion of radioactivity from potential NPP accidents [for] emergency response planning on national and international levels.

Fukushima blew up in early March 2011, when there was an anomalously cold weather pattern in the US. Places like Florida, which are usually warm, instead were hit pretty hard, especially with iodine-131.

I started writing this paper in February 2011, before Fukushima started. It dealt with how cold weather concentrates toxins in the air, and this affects symptoms of fibromyalgia sufferers. I subsequently discovered some research, mostly by Japanese scientists, who observed that fallout from the ’60s bomb tests increase in cold weather.

Actually it’s not just when it’s cold at the surface. It’s the average temperature between the surface and 18,000 feet. This is highly correlated with the 500 mb atmospheric height. When air cools down, it takes up less space. The 500 mb atmospheric height (HT500) is the point where the weight of the air from a certain altitude to the surface is constant.

When there is less space for the air, the concentration of toxins increases. This causes increased symptom ratings for fibromyalgia patients. This works for ME (myalgic encephalomyelitis) too. I got a horrible relapse just before New Year’s when it got really cold, and it shows no signs of departing.

But when the air is hot, toxins increase also. This is because the air is stagnant, and local sources of pollution like chemical plant emissions and auto exhaust tend to linger, and descend to the ground. There is also a health effect in hot weather. This works for ME too.

It turns out that toxins that originate from distant sources (like Japan) tend to increase in cold weather, while locally produced toxins increase in hot weather. Which one affects your health depends on the individual.

Miyake et al. (1962): “It was found that the specific radioactivity in rain water or the air activity was much higher when there was a trough at the 500-mb level [low HT500] or above and the core of a jet stream was located above or a little south of Tokyo.”

Miyake et al. (1960): “There is little correlation between the surface weather conditions and fallout while a considerably higher correlation was found among a trough at 500 mb, position of jet stream and air activity… These facts will account for the increase of the concentration of radioactive debris in the air and the rain with the passage of a trough line at 500 mb across an observation point.”

Chen et al. (1970): ”The dates of occurrence of peak concentrations of fallout particles generally coincided with (a) the arrival times of air masses at 500 mb and/or 300 mb after completing a cycle around the world, and (b) the passage of 500 mb troughs at Fayetteville… All these peaks have a direct correlation with the passage of the 500 mb trough. The dynamic explanation of this process is that to the immediate west of the upper-level trough, we usually find low-level divergence and upper-level convergence with the descending motion. It is this descending motion that brings down upper air and thus tends to increase the particle concentration. Miyake et al. (1960) also reported that similar meteorological conditions play an important role in the transport of radioisotopes from the stratosphere to the troposphere. They noted that the Sr-90 concentration in the ground-level air showed an increase after the passage of a 500 mb trough.”

Here is the forecast for HT500 at 1 PM on Friday:

This means that HT500 500 mb heights at that time will be low (cold) for Pennsylvania & Maryland, and also southern California. They are high for Montana and South Dakota. See if you can correlate it to your own health.

This is really cool. It’s a novel kind of cluster analysis technique I invented for the occasion:

This means that there are 3 groups of symptoms that move in tandem with respect to the weather conditions.

1. Pain and stiffness
2. Fatigue, concentration problems, memory problems, and sleep issues
3. Anxiety, depression, and gastrointestinal problems

All 3 might get worse when it’s cold. Or pain & stiffness will get worse, while fatigue, etc. get better. Or anxiety etc. might be worse when it’s hot. It depends on the individual and the particular toxin he or she is sensitive to.

I later associated it with beta radiation levels from the EPA data.

So those strange symptoms you are dealing with might be a result of nuclear fallout, as well as a host of other toxins.